By Jeffrey L Cummings, Serge Gauthier
The editors and participants deal with modern subject matters during this region via comprehensively referenced chapters, according to scientific adventure and clinical facts designed to stimulate dialogue of the place present remedy is heading.
content material: booklet hide; identify; Contents; members; Preface; Amyloid processing in Alzheimer's sickness mind; Cerebral amyloid (congophilic) angiopathy; medical use of neuroimaging within the assessment of dementia; scientific trials in gentle cognitive impairment; Muscarinic agonists in Alzheimer's illness; melancholy in dementia; melancholy in neurodegenerative issues and comparable stipulations; Use of antipsychotic medications in dementia; Assessing competency in Alzheimer's sickness: therapy consent capability and fiscal ability; administration of past due level dementia; Index.
summary: The editors and participants handle modern themes during this region via comprehensively referenced chapters, in line with scientific adventure and medical facts designed to stimulate dialogue of the place present remedy is heading
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Additional resources for Alzheimer's disease and related disorders annual. / 2001
73. 74. 75. 76. 77. 78. 79. Neuropathol Exp Neurol 1986; 45:79–90. Rengachary SS, Racela LS, Watanabe I, Abdou N. Neurosurgical and immunological implications of primary cerebral amyloid (Congophilic) angiopathy. Neurosurgery 1980; 7:1–9. Corsellis JAN, Brierley JB. An unusual type of pre-senile dementia (atypical Alzheimer’s disease with amyloid vascular changes). Brain 1954; 77:571–586. Dubas F, Gray F, Roullet E, Escourolle R. Leucoencéphalopathies artériopathiques (17 cas anatomo-cliniques).
Stroke 2000; 31:534–538. Eisenhauer PB, Johnson RJ, Wells JM, et al. Toxicity of various amyloid beta peptide species in cultured human blood–brain barrier endothelial cells: increased toxicity of Dutch-type mutant. J Neurosci Res 2000; 60:804–810. Weller RO, Massey A, Kuo Y-M, Roher AE. Cerebral amyloid angiopathy: accumulation of Abeta in interstitial ﬂuid drainage pathways in Alzheimer’s disease. 93. 94. 95. 96. 97. Ann NY Acad Sci 2000; 903:110–117. Van Dorpe J, Smeijers L, Dewachter I, et al.
There are no universally accepted criteria with which to grade the extent of amyloid-related vasculopathy. CAA may be absent in one brain region, while severe in another. 30 Usually the transition between affected and preserved vascular segments is abrupt. With increasing amyloid accumulation, the lengths of vascular segments that appear free of amyloid become shorter and their number decreases. The regional variation in CAA should be considered in sampling tissue for microscopic evaluation, or for planning a brain biopsy, although such an investigation is almost never performed strictly to evaluate severity of amyloid angiopathy.